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Protein S -glutathionylation can either activate or inhibit the activity and function of a range of structural proteins and enzymes [27,28]

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These include experimental models in rats such as acetaminophen hepatitis [36] and hepatic ischaemia/reperfusion injury, where the expression of inflammatory mediators (cyclooxygenase-1 (COX-2), tumour necrosis factor- (TNF-, interleukin (IL)-6, and monocyte chemoattractant protein-1 (MCP-1)) and reactive oxygen species (ROS) production have been demonstrated to be inhibited